New research published in the Journal of Clinical Endocrinology
& Metabolism has found “hedonic
hunger" or the desire to eat for taste and pleasure, triggers endogenous
reward signals in the brain that can prompt a person to eat more of the pleasurable
food—a phenomenon ultimately affects body mass and may be a factor in the
continuing rise of obesity.
Researchers at the University of Naples suggest endogenous
substances regulating reward mechanisms like the hormone ghrelin and chemical
compounds, such as 2-arachidonoylglycerol (2-AG), are involved.
For the study, the researchers assessed eight satiated healthy
adults, aged 21 to 33 years, feeding them each their personal favorite food
and, later, a less-palatable food of equal caloric and nutrient value.
Participants’ 2-AG and ghrelin levels were measured periodically. Plasma levels
of ghrelin and 2-AG increased during hedonic eating, with the favorite foods,
but not with non-hedonic eating. This increase suggests an activation of the
chemical reward system, which overrides the body’s signal that enough has been
eaten to restore energy.
“Hedonic hunger may powerfully stimulate overeating in an
environment where highly palatable foods are omnipresent, and contribute to the
surge in obesity," they said. “Understanding the physiological mechanisms
underlying this eating behavior may shed some light on the obesity epidemic.
Further research should confirm and extend our results to patients with obesity
or with other eating disorders in order to better understand the phenomenon of
hedonic eating."
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